How to Prevent a Cerebral Infarction - Oren Zarif - Cerebral Infarction
Cerebral infarction, or ischemic stroke, occurs when blood flow to the brain is interrupted or when there is a problem with the blood vessels that supply the brain. Lack of blood flow causes brain cells to die, resulting in a persistent focal neurologic deficit. The symptoms of cerebral infarction may be mild or severe, and if left untreated, can lead to permanent brain damage. There are several ways to prevent a cerebral infarction and protect yourself against it.
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The most common etiologies of cerebral infarction in young adults were cardiac embolism, hematologic causes, and lacunar stroke. Nearly one-third of first strokes had no apparent cause. However, the frequency of other vascular risk factors were associated with the occurrence of cerebral infarction in young adults. Although this study only included a small number of cases, it shows the potential association between the risk of developing cerebral infarction and several risk factors.
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Most cases of cerebral infarction are associated with high blood pressure and generalized hypotension, but other causes such as stroke or tumors can also cause the disease. The symptoms of cerebral infarction vary based on where in the brain the stroke occurs, and they may include weakness on one side of the body, loss of sensation on the other side of the body, and abnormal pupil dilation and reaction to light. If the stroke affects the left side of the brain, the patient may experience slurred speech, and reflexes may be affected.
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The treatments for acute cerebral infarction include mechanical thrombolysis, anticoagulation therapy, and surgery. Mechanical thrombectomy is not usually used in the case of acute massive cerebral infarction. Nimodipine is a selective Ca2+ antagonist that crosses the blood-brain barrier and acts on intracranial blood vessels. It is an accepted neuroprotective agent and is currently being studied for its safety.
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Cerebral infarction is a medical emergency that can be life-threatening. In most cases, it occurs when the blood flow to the brain is interrupted. A patient must have a high blood supply or risk permanent brain damage. If you suspect a stroke, consult a physician immediately. A lack of oxygen will increase the risk of brain damage. It is important to determine the exact cause of your stroke so that you can make an informed decision.
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Acute infarction may occur for several reasons. Some causes include arteriosclerotic cerebrovascular disease, arterial occlusion, and internal cartoid anomaly. In addition, thrombus from aneurysms may embolize into the brain, causing cerebral infarction. Another cause for cerebral infarction is the abnormal endothelium surrounding the brain. It is also important to consider how the thrombus in the brain formed.
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During the initial few hours after a cerebral infarction, neuroimaging is used to rule out the presence of an intracerebral hemorrhage, a tumor, or other rapidly growing tissue. CT can provide subtle evidence of a large anterior ischemic stroke. Evidence may include effacement of the insular cortical ribbon, loss of the gray-white junction, and a dense middle cerebral artery sign. Medium-sized infarcts can be detected as hypodensities, while small infarcts may be seen only with diffusion-weighted MRI.
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In a study of 316 patients, 212 (49.5%) had a probable cause, 80 (18.7%) had a possible cause, and 136 (31.8%) were without a diagnosis. The most common causes of first-time stroke were cardiac embolism, hematologic diseases, small vessel disease, and large artery atherosclerosis. Among asymptomatic stroke patients, only one of them had a recurrent stroke, and most of these were diagnosed with atrial fibrillation.
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Chronic meningitis can also cause a cerebral infarction. Infection with bacterial meningitis is a common cause, as are chronic infections of the meninges. The mortality rate for these strokes is high, and treatment should focus on early detection and intervention. Early diagnosis is crucial to maximize the chances of a good outcome and to prevent further cerebral ischaemia. Acute cerebral infarction may lead to seizure activity or a wide range of neurological complications.
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